Bilateral central scotomata due to intracranial tumour.
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Journal: 1984/August - British Journal of Ophthalmology
ISSN: 0007-1161
PUBMED: 6733068
Abstract:
Bilateral centrocaecal scotomata have been recognised as a sign of intrinsic optic nerve disease, usually associated with hereditary optic neuropathy, and nutritional or toxic amblyopias. This report describes four patients with central scotomata due to intracranial masses, three of whom recovered after surgical intervention. The clinician should be alerted to the association in patients with headaches, other neurological signs, and central visual loss.
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Br J Ophthalmol 68(7): 449-457
PMID: 6733068

Bilateral central scotomata due to intracranial tumour.

Abstract

Bilateral centrocaecal scotomata have been recognised as a sign of intrinsic optic nerve disease, usually associated with hereditary optic neuropathy, and nutritional or toxic amblyopias. This report describes four patients with central scotomata due to intracranial masses, three of whom recovered after surgical intervention. The clinician should be alerted to the association in patients with headaches, other neurological signs, and central visual loss.

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Selected References

These references are in PubMed. This may not be the complete list of references from this article.
  • Harris JR, Levene MB. Visual complications following irradiation for pituitary adenomas and craniopharyngiomas. Radiology. 1976 Jul;120(1):167–171. [PubMed] [Google Scholar]
  • HIRSCH O, HAMLIN H. Fate of visual fields and optic discs in pituitary tumors. Am J Ophthalmol. 1954 Jun;37(6):880–885. [PubMed] [Google Scholar]
  • RUCKER CW, KEARNS TP. Mistaken diagnoses in some cases of meningioma. Clinics in perimetry No. 5. Am J Ophthalmol. 1961 Jan;51:15–19. [PubMed] [Google Scholar]
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Abstract
Bilateral centrocaecal scotomata have been recognised as a sign of intrinsic optic nerve disease, usually associated with hereditary optic neuropathy, and nutritional or toxic amblyopias. This report describes four patients with central scotomata due to intracranial masses, three of whom recovered after surgical intervention. The clinician should be alerted to the association in patients with headaches, other neurological signs, and central visual loss.
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