IFN-gamma, IGIF, and IDDM.
Journal: 1997/March - Journal of Clinical Investigation
ISSN: 0021-9738
Relations:
Content
Citations
(3)
References
(6)
Diseases
(1)
Chemicals
(4)
Organisms
(6)
Processes
(2)
Anatomy
(2)
Similar articles
Articles by the same authors
Discussion board
J Clin Invest 99(3): 371-372

IFN-gamma, IGIF, and IDDM.

Full Text

The Full Text of this article is available as a PDF (74K).

Selected References

These references are in PubMed. This may not be the complete list of references from this article.
  • Lee MS, von Herrath M, Reiser H, Oldstone MB, Sarvetnick N. Sensitization to self (virus) antigen by in situ expression of murine interferon-gamma. J Clin Invest. 1995 Feb;95(2):486–492.[PMC free article] [PubMed] [Google Scholar]
  • Rothe H, Jenkins NA, Copeland NG, Kolb H. Active stage of autoimmune diabetes is associated with the expression of a novel cytokine, IGIF, which is located near Idd2. J Clin Invest. 1997 Feb 1;99(3):469–474.[PMC free article] [PubMed] [Google Scholar]
  • Okamura H, Tsutsi H, Komatsu T, Yutsudo M, Hakura A, Tanimoto T, Torigoe K, Okura T, Nukada Y, Hattori K, et al. Cloning of a new cytokine that induces IFN-gamma production by T cells. Nature. 1995 Nov 2;378(6552):88–91. [PubMed] [Google Scholar]
  • Ushio S, Namba M, Okura T, Hattori K, Nukada Y, Akita K, Tanabe F, Konishi K, Micallef M, Fujii M, et al. Cloning of the cDNA for human IFN-gamma-inducing factor, expression in Escherichia coli, and studies on the biologic activities of the protein. J Immunol. 1996 Jun 1;156(11):4274–4279. [PubMed] [Google Scholar]
  • Wicker LS, Todd JA, Peterson LB. Genetic control of autoimmune diabetes in the NOD mouse. Annu Rev Immunol. 1995;13:179–200. [PubMed] [Google Scholar]
  • Hultgren B, Huang X, Dybdal N, Stewart TA. Genetic absence of gamma-interferon delays but does not prevent diabetes in NOD mice. Diabetes. 1996 Jun;45(6):812–817. [PubMed] [Google Scholar]
Full Text
Selected References
Collaboration tool especially designed for Life Science professionals.Drag-and-drop any entity to your messages.